Difference between revisions of "Team:HSNU-TAIPEI/projectmercury"

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<h1>Project<span>Mercury</span></h1>
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<div class="section">
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  <article class="article">
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    <h3 class="article-title">Introduction</h3>
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    <ol class="article-ol">
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      <li>
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        <span>Why do we detect mercury?</span>
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        <p class="article-p">通常食用油中是不會含有如此超標的重金屬汞。食用油裡會含有超標的汞原因是動物攝入甲基汞後囤積於內臟(註1),而餿水油是由內臟油加工製成,但汞不容易被人體代謝掉進而累積在體內造成嚴重的疾病,所以我們才會偵測汞這個重金屬。</p>
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      </li>
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      <li>
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        <span>The harm of mercury</span>
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        <p class="article-p">甲基汞是已知的神經毒物,會抑制神經細胞的分裂及電離子的移動並干擾發育中的腦部結構,除此之外,甲基汞還能引起心血管疾病包括心肌梗塞與缺血性心臟病、高血壓及心律不整(註2)。</p>
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      </li>
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      <li>
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        <span>Regulation</span>
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        <ul class="article-ul">
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          <li>用油脂類衛生標準第二條:重金屬及芥酸之最大容許量:汞0.05 ppm</li>
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          <li>食米重金屬限量標準第二條:食米中重金屬含量應符合以下限量:0.05ppm</li>
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        </ul>
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      </li>
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    </ol>
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  </article>
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  <article class="article">
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    <h3 class="article-title">Circuit Design</h3>
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    <div class="article-img"><img src="https://static.igem.org/mediawiki/2015/7/7e/2015hsnu-mercury1.png"></div>
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    <p class="article-p">MerR可以和汞結合然後改變DNA的結構好讓RNA聚合酶方便轉入。PmerT可以被MerR控制。PmerT步行進行轉錄是因為MerR會改變PmerT的形狀。總而言之,只要汞與MerR結合,後續反應就可進行。(註3)(註4)</p>
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    <div class="article-img"><img src="https://static.igem.org/mediawiki/2015/b/b2/2015hsnu-mercury_2.png"></div>
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  </article>
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  <article class="article">
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    <h3 class="article-title">Result</h3>
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  </article>
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  <article class="article">
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    <h3 class="article-title">Reference</h3>
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    <ul class="article-ul">
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      <li>(註1)Metabolism of mercury, administered as methylmercuric chloride or mercuric chloride, by lactating ruminants.</li>
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      <li>(註2)台北醫學大學公共衛生學系碩士班碩士論文Graduate Institute.</li>
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      <li>(註3)Brown, N. L., J. V. Stoyanov, et al. (2003). "The MerR family of transcriptional regulators." FEMS Microbiol Rev 27(2-3): 145-163.</li>
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      <li>(註4)Park, S. J., J. Wireman, et al. (1992). "Genetic analysis of the Tn21 mer operator-promoter." J Bacteriol 174(7): 2160-2171.</li>
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    </ul>
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  </article>
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Revision as of 13:51, 3 September 2015

ProjectMercury

Introduction

  1. Why do we detect mercury?

    通常食用油中是不會含有如此超標的重金屬汞。食用油裡會含有超標的汞原因是動物攝入甲基汞後囤積於內臟(註1),而餿水油是由內臟油加工製成,但汞不容易被人體代謝掉進而累積在體內造成嚴重的疾病,所以我們才會偵測汞這個重金屬。

  2. The harm of mercury

    甲基汞是已知的神經毒物,會抑制神經細胞的分裂及電離子的移動並干擾發育中的腦部結構,除此之外,甲基汞還能引起心血管疾病包括心肌梗塞與缺血性心臟病、高血壓及心律不整(註2)。

  3. Regulation
    • 用油脂類衛生標準第二條:重金屬及芥酸之最大容許量:汞0.05 ppm
    • 食米重金屬限量標準第二條:食米中重金屬含量應符合以下限量:0.05ppm

Circuit Design

MerR可以和汞結合然後改變DNA的結構好讓RNA聚合酶方便轉入。PmerT可以被MerR控制。PmerT步行進行轉錄是因為MerR會改變PmerT的形狀。總而言之,只要汞與MerR結合,後續反應就可進行。(註3)(註4)

Result

Reference

  • (註1)Metabolism of mercury, administered as methylmercuric chloride or mercuric chloride, by lactating ruminants.
  • (註2)台北醫學大學公共衛生學系碩士班碩士論文Graduate Institute.
  • (註3)Brown, N. L., J. V. Stoyanov, et al. (2003). "The MerR family of transcriptional regulators." FEMS Microbiol Rev 27(2-3): 145-163.
  • (註4)Park, S. J., J. Wireman, et al. (1992). "Genetic analysis of the Tn21 mer operator-promoter." J Bacteriol 174(7): 2160-2171.