Difference between revisions of "Team:Marburg/Minicells"
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<div style="position:relative;display:inline-block;padding:15px;background:#F8F8F8;border-style:solid;border-color:#FF8F45;border-width:2px;border-radius:10px;text-align:justify;font-size:13pt;line-height:150%;margin:10px;margin-bottom:30px;"> | <div style="position:relative;display:inline-block;padding:15px;background:#F8F8F8;border-style:solid;border-color:#FF8F45;border-width:2px;border-radius:10px;text-align:justify;font-size:13pt;line-height:150%;margin:10px;margin-bottom:30px;"> | ||
<h1 >Background</h1> | <h1 >Background</h1> | ||
| − | <p>Bacterial minicells are produced by asymmetric cell division, which is caused by a knock out of the minCDE-system. Naturally, this system regulates symmetric cell division.[1] The Min-proteins bind to the cell poles and oscillate between them. MinC functions as inhibitor for FtsZ, which forms the Z-ring and ultimately initiates division of the cell. This symmetric division ensures that each cell gets a copy of the chromosome and all other cell components and thus, is able to live on its own. If this system is disturbed, cell division occurs randomly leading to the formation of minicells.</p> | + | <p>Bacterial minicells are produced by asymmetric cell division, which is caused by a knock out of the minCDE-system. Naturally, this system regulates symmetric cell division.[1] The Min-proteins bind to the cell poles and oscillate between them. MinC functions as inhibitor for FtsZ, which forms the Z-ring and ultimately initiates division of the cell. This symmetric division ensures that each cell gets a copy of the chromosome and all other cell components and thus, is able to live on its own. If this system is disturbed, cell division occurs randomly leading to the formation of minicells. |
| + | <figure style="text-align:center;"> | ||
| + | <img src="https://static.igem.org/mediawiki/2015/e/e0/MR_pic_minsystem.png" width="400px" alt="minsystem" /> | ||
| + | <figcaption style="margin-top:5px;font-size:11pt;color:#606060;text-align:justify;line-height:110%"><b>Figure X:</b> Normal functional min-system </figcaption> | ||
| + | </figure> | ||
| + | </p> | ||
</div> | </div> | ||
Revision as of 21:14, 17 September 2015
Aim
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Project Design
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Results
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Outlook
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Background
Bacterial minicells are produced by asymmetric cell division, which is caused by a knock out of the minCDE-system. Naturally, this system regulates symmetric cell division.[1] The Min-proteins bind to the cell poles and oscillate between them. MinC functions as inhibitor for FtsZ, which forms the Z-ring and ultimately initiates division of the cell. This symmetric division ensures that each cell gets a copy of the chromosome and all other cell components and thus, is able to live on its own. If this system is disturbed, cell division occurs randomly leading to the formation of minicells.
